Dr. Betty Martini, D.Hum.
Mission Possible World Health International
9270 River Club Parkway
Duluth, Georgia 30097
Telephone: 770-242-2599
E-Mail: BettyM19@mindspring.com

Posted: 26 February 2009

I just received this study from Dr. Russell Blaylock with these words: "This is enough reason to ban aspartame". How much more evidence could anyone need with study after study showing how deadly this addictive excitoneurotoxic carcinogenic drug is. This combined with the fact that the FDA themselves revoked the petition for approval and it would never have gotten on the market without the political chicanery of Don Rumsfeld. http://www.soundandfury.tv/pages/rumsfeld2.html

The Trocho Study shows the formaldehyde from the free methyl alcohol embalms living tissue and damages DNA. When you damage DNA you can destroy humanity. The Ramazzini Studies show aspartame is a multipotential carcinogen even in small amounts and can be passed on from the mother. This violates the Delaney Amendment and FDA toxicologist, Dr. Adrian Gross, told Congress aspartame violated this amendment because it also causes brain tumors. What does it take to get this deadly chemical poison off the market. Consider that aspartame damages the mitochondria and interacts with virtually all drugs and vaccines. How can an FDA who cares at all about safe food and drugs allow this on the market and turn their back even on laws such as the Citizen Petition to Ban case when hasn't been answered for over 6 years. The law says it must be answered in 180 days. The imminent health hazard amendment that requires answering in a week to ten days was sent over a year ago.

We're at the point to either ban aspartame or ban the FDA, one or the other. Without the FDA protecting the aspartame manufacturers it would have taken off the market years ago.

Dr. Betty Martini, D.Hum.
Founder, Mission Possible World Health International
9270 River Club Parkway
Duluth, Georgia 30097
E-Mail: BettyM19@mindspring.com

Aspartame Toxicity Center: http://www.holisticmed.com/aspartame


Medical Text: Aspartame Disease: An Ignored Epidemic, http://www.sunsentpress.com, H. J. Roberts, M.D.

Excitotoxins: The Taste That Kills, neurosurgeon Russell Blaylock, M.D., http://www.russellblaylockmd.com

Aspartame Documentary: "Sweet Misery: A Poisoned World" : http://www.soundandfury.tv

Daily Consumption of Diet Soda Linked to Metabolic Syndrome, Type 2 Diabetes

News Author: Laurie Barclay, MD
CME Author: Laurie Barclay, MD
Release Date: February 11, 2009; Valid for credit through February 11, 2010
Credits Available
Physicians - maximum of 0.25 AMA PRA Category 1 Credit(s)(tm) for physicians;
Family Physicians - up to 0.25 AAFP Prescribed credit(s) for physicians;
Nurses - 0.25 ANCC contact hours (None of these credits is in the area of pharmacology)

To participate in this internet activity: (1) review the target audience, learning objectives, and author disclosures; (2) study the education content; (3) take the post-test and/or complete the evaluation; (4) view/print certificate View details.

Learning Objectives

Upon completion of this activity, participants will be able to:

  1. Describe the association between diet soda consumption and the risk for incident metabolic syndrome and its components.
  2. Describe the association between diet soda consumption and the risk for incident type 2 diabetes.

Authors and Disclosures

Laurie Barclay, MD
Disclosure: Laurie Barclay, MD, has disclosed no relevant financial relationships.

Laurie Scudder, MS, NP
Disclosure: Laurie Scudder, MS, NP, has disclosed no relevant financial information.

Brande Nicole Martin
Disclosure: Brande Nicole Martin has disclosed no relevant financial information.

February 11, 2009 - Drinking diet soda at least daily is associated with significantly greater risks for select incident components of the metabolic syndrome (MetSyn) and type 2 diabetes, according to the results of an observational study reported in the January 16 Online First issue of Diabetes Care.

"Two longitudinal cohort studies have shown positive associations between diet soda consumption and incident MetSyn independent of baseline measures of adiposity," write Jennifer A. Nettleton, PhD, from the University of Texas Health Sciences Center in Houston, and colleagues. "Replication of previously observed diet soda-MetSyn associations in a distinct cohort would bolster their credibility and provide further insight into the nature of the relationship. Previous studies have not addressed associations between diet soda and individual MetSyn components or risk of type 2 diabetes nor have they fully addressed potential longitudinal mediators of these relationships, i.e., changes in adiposity status."

The goal of this study was to evaluate associations between diet soda consumption and the risk for incident MetSyn, its components, and type 2 diabetes in the Multi-Ethnic Study of Atherosclerosis (MESA).

Initial evaluation was performed from 2000 to 2002, at which time baseline food frequency questionnaires measured diet soda consumption. Three follow-up evaluations were performed from 2002 to 2003, 2004 to 2005, and 2005 to 2007. Incident type 2 diabetes was defined as fasting glucose levels of more than 126 mg/dL, self-reported type 2 diabetes, or use of diabetes medication. National Cholesterol Education Program Adult Treatment Panel 3 criteria were used to define MetSyn and its components. After adjustment for demographic, lifestyle, and dietary confounders, hazard ratios (HRs) were estimated for type 2 diabetes, MetSyn, and MetSyn components.

Compared with participants who did not drink diet soda, those who drank diet soda at least daily had a 36% greater relative risk for incident MetSyn (HR, 1.36; 95% confidence interval [CI], 1.11 - 1.66) and a 67% greater relative risk for incident type 2 diabetes (HR, 1.67; 95% CI, 1.27 - 2.20).

Of the individual components of MetSyn, only high waist circumference (men: = 102 cm; women: = 88 cm) and high fasting glucose levels (= 100 mg/dL) were prospectively associated with consumption of diet soda. Associations between diet soda intake and type 2 diabetes were independent of baseline measures of adiposity or changes in these measures. In contrast, associations between diet soda and MetSyn were not independent of these factors.

"Although these observational data cannot establish causality, consumption of diet soda at least daily was associated with significantly greater risks of select incident MetSyn components and type 2 diabetes," the study authors write.

Limitations of this study include observational design, precluding determination of causality; possible confounding by other dietary and lifestyle/behavioral factors; and difficulties in estimating intake of diet soda or artificial sweetener.

"These results corroborate findings from the ARIC [Atherosclerosis Risk in Communities] and Framingham studies and show stronger adverse associations exist between diet soda and type 2 diabetes," the study authors conclude. "Diet soda consumption, either independently or in conjunction with other dietary and lifestyle behaviors, may lead to weight gain, impaired glucose control, and eventual diabetes."

The National Heart, Lung, and Blood Institute supported this study. The study authors have disclosed no relevant financial relationships.

Diabetes Care. Published online January 16, 2009.
Clinical Context

Diet soda and other artificially sweetened beverages were originally considered "benign" because they contribute no calories and few nutrients to the diet. Previously reported links between intake of diet soda and MetSyn were therefore thought to result from residual confounding by other dietary, lifestyle, or demographic factors.

However, biological mechanisms possibly underlying these associations may be plausible, such as artificial sweeteners increasing the desire for and consumption of energy-dense foods or interfering with the ability to accurately estimate energy intake and remaining energy needs. Better understanding of these associations has important implications for nutritional counseling.
Study Highlights